https://nova.newcastle.edu.au/vital/access/ /manager/Index en-au 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:34799 Wed 27 Apr 2022 14:45:47 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:27794 Wed 22 May 2019 14:45:58 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:43392 Wed 22 Mar 2023 15:32:42 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30305 d = −0.82), while questioning skills (d = 0.56) decreased. There were no significant differences in any other skills grouping, although checking, information organization, and empathic communication skills tended to increase post-training. A dose effect was observed for agenda setting, with trainees who attended more CST sessions outperforming those attending fewer. Conclusion: Findings support the generalization and translation of ComPsych CST to psychiatry.]]> Wed 18 Oct 2017 12:14:52 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:43186 Wed 14 Sep 2022 08:51:31 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:21216 Wed 11 Apr 2018 18:06:30 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:29013 P < 2 × 10⁻¹⁶). Further, the gene set analyses revealed several miRNAs regulating schizophrenia risk genes, with the strongest enrichment for targets of miR-9-5p (P = .0056 for enrichment among the top 1% most-associated single-nucleotide polymorphisms, corrected for multiple testing). It is further of note that MIR9-2 is located in a genomic region showing strong evidence for association with schizophrenia (P = 7.1 × 10⁻⁸). The second and third strongest gene set signals were seen for the targets of miR-485-5p and miR-137, respectively. Conclusions and Relevance: This study provides evidence for a role of miR-9-5p in the etiology of schizophrenia. Its implication is of particular interest as the functions of this neurodevelopmental miRNA tie in with established disease biology: it has a regulatory loop with the fragile X mental retardation homologue FXR1 and regulates dopamine D₂ receptor density.]]> Wed 11 Apr 2018 15:28:50 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:22665 Wed 11 Apr 2018 12:37:33 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:49007 Wed 03 May 2023 12:17:36 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:49885 Tue 13 Jun 2023 13:08:41 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:33388 Tue 03 Sep 2019 17:54:11 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:40024 Thu 21 Jul 2022 09:47:00 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:38960 Thu 17 Mar 2022 13:04:30 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26878 Thu 16 Mar 2017 14:51:42 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:34615 Thu 04 Apr 2019 09:04:20 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:9528 Sat 24 Mar 2018 08:35:32 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:10415 Sat 24 Mar 2018 08:12:38 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:12278 Sat 24 Mar 2018 08:10:09 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:18309 Sat 24 Mar 2018 08:04:40 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20699 Sat 24 Mar 2018 07:55:40 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:29862 2 = 1.1E-03, P = 4.1E-04), reflecting the published relationship between maternal age and SCZ risk in offspring by McGrath et al in 2014. Conclusions and Relevance: This study provides evidence for a significant overlap between genetic factors associated with risk of SCZ and genetic factors associated with AFB. It has been reported that SCZ risk associated with increased maternal age is explained by the age of the father and that de novo mutations that occur more frequently in the germline of older men are the underlying causal mechanism. This explanation may need to be revised if, as suggested herein and if replicated in future studies, there is also increased genetic risk of SCZ in older mothers.]]> Sat 24 Mar 2018 07:40:47 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30942 Sat 24 Mar 2018 07:33:42 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:25725 Sat 24 Mar 2018 07:33:29 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30050 Sat 24 Mar 2018 07:31:15 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26938 Sat 24 Mar 2018 07:27:30 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:25351 Sat 24 Mar 2018 07:24:42 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:23470 Sat 24 Mar 2018 07:13:01 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:44131 Sat 08 Oct 2022 12:36:32 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:48473 Fri 17 Mar 2023 17:56:40 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:49165 Fri 05 May 2023 15:30:14 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:49108 Fri 05 May 2023 11:32:00 AEST ]]>